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Dyslexia
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Dyslexia
Synonyms
Reading disorder, alexia
An example of OpenDyslexic typeface, used to try to help with common reading errors in dyslexia.[1]
Specialty
Neurology, pediatrics
Symptoms
Trouble reading[2]
Usual onset
School age[3]
Causes
Genetic and environmental factors[3]
Risk factors
Family history, attention deficit hyperactivity disorder[4]
Diagnostic method
Series memory, spelling, vision, and reading test[5]
Similar conditions
Hearing or vision problems, insufficient teaching[3]
Treatment
Adjusting teaching methods[2]
Frequency
3–7% [3][6]
[edit on Wikidata]
Dyslexia, also known as reading disorder, is characterized by trouble with reading despite normal intelligence.[2][7] Different people are affected to varying degrees.[4] Problems may include difficulties in spelling words, reading quickly, writing words, "sounding out" words in the head, pronouncing words when reading aloud and understanding what one reads.[4][8] Often these difficulties are first noticed at school.[3] When someone who previously could read loses their ability, it is known as alexia.[4] The difficulties are involuntary and people with this disorder have a normal desire to learn.[4]
Dyslexia is believed to be caused by both genetic and environmental factors.[3] Some cases run in families.[4] It often occurs in people with attention deficit hyperactivity disorder (ADHD) and is associated with similar difficulties with numbers.[3] It may begin in adulthood as the result of a traumatic brain injury, stroke, or dementia.[2] The underlying mechanisms of dyslexia are problems within the brain's language processing.[4] Dyslexia is diagnosed through a series of tests of memory, spelling, vision, and reading skills.[5] Dyslexia is separate from reading difficulties caused by hearing or vision problems or by insufficient teaching.[3]
Treatment involves adjusting teaching methods to meet the person's needs.[2] While not curing the underlying problem, it may decrease the degree of symptoms.[9] Treatments targeting vision are not effective.[10] Dyslexia is the most common learning disability and occurs in all areas of the world.[3][11] It affects 3–7% of the population,[3][6] however, up to 20% may have some degree of symptoms.[12] While dyslexia is more often diagnosed in men,[3] it has been suggested that it affects men and women equally.[11] Some believe that dyslexia should be best considered as a different way of learning, with both benefits and downsides.[13][14]
Contents
[hide]
1 Classification
1.1 Definition
2 Signs and symptoms
2.1 Language
2.2 Associated conditions
3 Causes
3.1 Neuroanatomy
3.2 Genetics
3.3 Gene–environment interaction
4 Mechanisms
5 Diagnosis
5.1 Central dyslexias
5.1.1 Surface dyslexia
5.1.2 Phonological dyslexia
5.1.3 Deep dyslexia
5.2 Peripheral dyslexias
5.2.1 Pure dyslexia
5.2.2 Hemianopic dyslexia
5.3 Neglect dyslexia
5.3.1 Attentional dyslexia
6 Management
7 Prognosis
8 Epidemiology
9 History
10 Research and society
11 See also
12 References
13 Further reading
14 External links
Classification
Dyslexia is thought to have two types of cause, one related to language processing and another to visual processing.[15] It is considered a cognitive disorder, not a problem with intelligence. However, emotional problems often arise because of it.[15] Some published definitions are purely descriptive, whereas others propose causes. The latter usually cover a variety of reading skills and deficits, and difficulties with distinct causes rather than a single condition.[16] The National Institute of Neurological Disorders and Stroke definition describes dyslexia as "difficulty with phonological processing (the manipulation of sounds), spelling, and/or rapid visual-verbal responding".[2] The British Dyslexia Association definition describes dyslexia as "a learning difficulty that primarily affects the skills involved in accurate and fluent word reading and spelling" and is characterized by "difficulties in phonological awareness, verbal memory and verbal processing speed".[17]
Acquired dyslexia or alexia may be caused by brain damage due to stroke or atrophy.[18][19] Forms of alexia include pure alexia, surface dyslexia, semantic dyslexia, phonological dyslexia, and deep dyslexia.[20]
Definition
There is some variability in the definition of dyslexia. Some sources, such as the U.S. National Institutes of Health, define it specifically as a learning disorder.[2] Other sources, however, define it simply as an inability to read in the context of normal intelligence, and distinguish between developmental dyslexia (a learning disorder) and acquired dyslexia (loss of the ability to read caused by brain damage). ICD 10, the manual of medical diagnosis used in much of the world, includes separate diagnoses for "developmental dyslexia" (81.0)[21] and for "dyslexia and alexia" (48.0).[22] DSM 5, the manual of psychiatric diagnosis used in the United States, does not specifically define dyslexia, justifying this decision by stating that "the many definitions of dyslexia and dyscalculia meant those terms would not be useful as disorder names or in the diagnostic criteria". Instead it includes dyslexia in a category called specific learning disorders.[23]
Signs and symptoms
See also: Characteristics of dyslexia
In early childhood, symptoms that correlate with a later diagnosis of dyslexia include delayed onset of speech, difficulty distinguishing left from right, difficulty with direction,[24] and a lack of phonological awareness, as well as being easily distracted by background noise.[10] A common myth closely associates dyslexia with mirror writing and reading letters or words backwards.[25] These behaviors are seen in many children as they learn to read and write, and are not considered to be defining characteristics of dyslexia.[10]
School-age children with dyslexia may exhibit signs of difficulty in identifying or generating rhyming words, or counting the number of syllables in words – both of which depend on phonological awareness.[26] They may also show difficulty in segmenting words into individual sounds or may blend sounds when producing words, indicating reduced phonemic awareness.[27] Difficulties with word retrieval or naming things is also associated with dyslexia.[28]:647 People with dyslexia are commonly poor spellers, a feature sometimes called dysorthographia or dysgraphia, which depends on orthographic coding.[10]
Problems persist into adolescence and adulthood and may accompany difficulties with summarizing stories, memorization, reading aloud, or learning foreign languages. Adults with dyslexia can often read with good comprehension, though they tend to read more slowly than others without a learning difficulty and perform worse in spelling tests or when reading nonsense words – a measure of phonological awareness.[29]
Language
Main article: Orthographies and dyslexia
The orthographic complexity of a language directly impacts how difficult learning to read the language is.[30]:266 English and French have comparatively "deep" phonemic orthographies within the Latin alphabet writing system, with complex structures employing spelling patterns on several levels: letter-sound correspondence, syllables, and morphemes.[31]:421 Languages such as Spanish, Italian and Finnish have mostly alphabetic orthographies, which primarily employ letter-sound correspondence – so-called shallow orthographies – which for dyslexics makes them easier to learn.[30]:266 Logographic writing systems, such as Chinese characters, have extensive symbol use, and pose problems for dyslexic learners.[32]
Associated conditions
Dyslexia is often accompanied by several learning disabilities, but it is unclear whether they share underlying neurological causes.[33] These associated disabilities include:
Dysgraphia – A disorder which primarily expresses itself through difficulties with writing or typing, but in some cases through difficulties associated with eye–hand coordination and direction or sequence-oriented processes such as tying knots or carrying out repetitive tasks.[34] In dyslexia, dysgraphia is often multifactorial, due to impaired letter-writing automaticity, organizational and elaborative difficulties, and impaired visual word forming which makes it more difficult to retrieve the visual picture of words required for spelling.[34]
Attention deficit hyperactivity disorder (ADHD) – A disorder characterized by problems paying attention, excessive activity, or taking action without forethought.[35] Dyslexia and ADHD commonly occur together.[6][36][37] Either 15%[10] or 12–24% of people with dyslexia have ADHD.[38] 35% of people with ADHD have dyslexia.[10]
Auditory processing disorder – A listening disability that affects the ability to process auditory information.[39][40] This can lead to problems with auditory memory and auditory sequencing. Many people with dyslexia have auditory processing problems, and may develop their own logographic cues to compensate for this type of deficit. Some research indicates that auditory processing skills could be the primary shortfall in dyslexia.[41][42]
Developmental coordination disorder – A neurological condition characterized by marked difficulty in carrying out routine tasks involving balance, fine-motor control, kinesthetic coordination, difficulty in the use of speech sounds, problems with short-term memory, and organization.[43]
Causes
Inferior parietal lobule (superior view). Some dyslexics demonstrate less electrical activation in this area.
Main article: Theories of dyslexia
Researchers have been trying to find the neurobiological basis of dyslexia since the condition was first identified in 1881.[44][45] For example, some have tried to associate the common problem among dyslexics of not being able to see letters clearly to abnormal development of their visual nerve cells.[46]
Neuroanatomy
Main article: Neurological research into dyslexia
Modern neuroimaging techniques such as functional magnetic resonance imaging (fMRI) and positron emission tomography (PET) have shown a correlation between both functional and structural differences in the brains of children with reading difficulties.[47] Some dyslexics show less electrical activation in parts of the left hemisphere of the brain involved with reading, such as the inferior frontal gyrus, inferior parietal lobule, and the middle and ventral temporal cortex.[41] Over the past decade, brain activation studies using PET to study language have produced a breakthrough in the understanding of the neural basis of language. Neural bases for the visual lexicon and for auditory verbal short-term memory components have been proposed,[48] with some implication that the observed neural manifestation of developmental dyslexia is task-specific (i.e. functional rather than structural). fMRIs in dyslexics have provided important data which point to the interactive role of the cerebellum and cerebral cortex as well as other brain structures.[49][50]
The cerebellar theory of dyslexia proposes that impairment of cerebellum-controlled muscle movement affects the formation of words by the tongue and facial muscles, resulting in the fluency problems that are characteristic of some dyslexics. The cerebellum is also involved in the automatization of some tasks, such as reading.[51] The fact that some dyslexic children have motor task and balance impairments has been used as evidence for a cerebellar role in their reading difficulties. However, the cerebellar theory is not supported by controlled research studies.[52]
Genetics
Main article: Genetic research into dyslexia
Research into potential genetic causes of dyslexia has its roots in post-autopsy examination of the brains of people with dyslexia.[46] Observed anatomical differences in the language centers of such brains include microscopic cortical malformations known as ectopias, more rarely, vascular micro-malformations, and microgyrus.[53] The previously cited studies and others[54] suggest that abnormal cortical development presumed to occur before or during the sixth month of fetal brain development was the cause of the abnormalities. Abnormal cell formations in dyslexics have also been reported in non-language cerebral and subcortical brain structures.[55] Several genes have been associated with dyslexia, including DCDC2 and KIAA0319 on chromosome 6,[56] and DYX1C1 on chromosome 15.[57]
Gene–environment interaction
Main article: Gene–environment interaction
The contribution of gene–environment interaction to reading disability has been intensely studied using twin studies, which estimate the proportion of variance associated with a person's environment and the proportion associated with their genes. Studies examining the influence of environmental factors such as parental education[58] and teacher quality[59] have determined that genetics have greater influence in supportive, rather than less optimal, environments.[60] However, more optimal conditions may just allow those genetic risk factors to account for more of the variance in outcome because the environmental risk factors have been minimized.[60] As environment plays a large role in learning and memory, it is likely that epigenetic modifications play an important role in reading ability. Animal experiments and measures of gene expression and methylation in the human periphery are used to study epigenetic processes; however, both types of study have many limitations in the extrapolation of results for application to the human brain.[61]
Mechanisms
Main article: Dual-route hypothesis to reading aloud
The dual-route theory of reading aloud was first described in the early 1970s.[62] This theory suggests that two separate mental mechanisms, or cognitive routes, are involved in reading aloud.[63] One mechanism is the lexical route, which is the process whereby skilled readers can recognize known words by sight alone, through a "dictionary" lookup procedure.[64] The other mechanism is the nonlexical or sublexical route, which is the process whereby the reader can "sound out" a written word.[64][65] This is done by identifying the word's constituent parts (letters, phonemes, graphemes) and applying knowledge of how these parts are associated with each other, for example, how a string of neighboring letters sound together.[62] The dual-route system could explain the different rates of dyslexia occurrence between different languages (e.g. the Spanish language dependence on phonological rules accounts for the fact that Spanish-speaking children show a higher level of performance in non-word reading, when compared to English-speakers).[30][66]
Dyslexia disorder is not caused by mutation in one gene; in fact, it appears to involve the combined effects of several genes. Studying the cognitive problems associated with other disorders helps to better understand the genotype-phenotype link of dyslexia.[67] Neurophysiological and imaging procedures are being used to ascertain phenotypic characteristics in dyslexics, thus identifying the effects of certain genes.[68]
Diagnosis
There are tests that can indicate with high probability whether a person is a dyslexic.[69] If diagnostic testing indicates that a person may be dyslexic, such tests are often followed up with a full diagnostic assessment to determine the extent and nature of the disorder.[70] Tests can be administered by a teacher or computer.[71] Some test results indicate how to carry out teaching strategies.[71][72]
Central dyslexias
Central dyslexias include surface dyslexia, semantic dyslexia, phonological dyslexia, and deep dyslexia.[18][73] ICD-10 reclassified the previous distinction between dyslexia (315.02 in ICD-9) and alexia (315.01 in ICD-9) into a single classification as R48.0. The terms are applied to developmental dyslexia and inherited dyslexia along with developmental aphasia and inherited alexia, which are considered synonymous.[74]
Surface dyslexia
Main article: Surface dyslexia
In surface dyslexia, words with regular pronunciations (highly consistent with their spelling, e.g. mint) are read more accurately than words with irregular pronunciation, such as colonel.[75] Difficulty distinguishing homophones is a diagnostic used for some forms of surface dyslexia. This disorder is usually accompanied by surface agraphia and fluent aphasia.[76] Acquired surface dyslexia arises when a previously literate person experiences brain damage, which results in pronunciation errors that indicate impairment of the lexical route.[18][77][78]
Phonological dyslexia
Broca's area – (lateral view) dyslexics overuse this area which is associated with speech.[79]
Main article: Phonological dyslexia
In phonological dyslexia, sufferers can read familiar words but have difficulty with unfamiliar words, such as invented pseudo-words.[80] Phonological dyslexia is associated with lesions in the parts of the brain supplied with blood by the middle cerebral artery. The superior temporal lobe is often also involved. Furthermore, dyslexics compensate by overusing a front-brain region called Broca's area, which is associated with aspects of language and speech.[81] The Lindamood Phoneme Sequencing Program (LiPS) is used to treat phonological dyslexia.[82] This system is based on a three-way sensory feedback process, using auditory, visual, and oral skills to learn to recognize words and word patterns. Case studies with a total of three patients found a significant improvement in spelling and reading ability after using LiPS.[83]
Deep dyslexia
See also: Deep dyslexia
Individuals with deep dyslexia experience both semantic paralexia (para-dyslexia) and phonological dyslexia, which causes the person to read a word and then say a related meaning instead of the denoted meaning.[84] Deep alexia is associated with clear phonological processing impairments.[18] Deep dyslexia is caused by widespread damage to the brain that often includes the left hemisphere.[85] The "continuum" hypothesis claims that deep dyslexia develops from phonological dyslexia.[86]
Peripheral dyslexias
Peripheral dyslexias have been described as affecting the visual analysis of letters as a result of brain injury.[87] Hemianopsia, a visual field loss on the left/right side of the vertical midline, is associated with this condition.[88][89]
Pure dyslexia
Main article: Pure alexia
Pure, or phonologically-based,[90] dyslexia, also known as agnosic dyslexia, dyslexia without agraphia, and pure word blindness, is dyslexia due to difficulty in recognizing written sequences of letters (such as words), or sometimes even letters. It is considered '"pure" because it is not accompanied by other significant language-related impairments. Pure dyslexia does not affect speech, handwriting style, language or comprehension impairments.[91] Pure dyslexia is caused by lesions on the visual word form area (VWFA). The VWFA is composed of the left lateral occipital sulcus and is activated during reading. A lesion in the VWFA stops transmission between the visual cortex and the left angular gyrus. It can also be caused by a lesion involving the left occipital lobe or the splenium. It is usually accompanied by a homonymous hemianopsia in the right side of the visual field.[87] Multiple oral re-reading (MOR) is a treatment for pure dyslexia.[92] It is considered a top-down processing technique in which affected individuals read and reread texts a predetermined number of times or until reading speed or accuracy improves a predetermined amount.[93]
Hemianopic dyslexia
Hemianopic dyslexia is commonly considered to derive from visual field loss due to damage to the primary visual cortex.[94] Sufferers may complain of abnormally slow reading but are able to read individual words normally. This is the most common form of peripheral alexia, and the form with the best evidence of effective treatments.[95]
Neglect dyslexia
In neglect dyslexia, some letters, most commonly those at the beginning or left side of a word, are skipped or misread during reading.[96] This alexia is associated with right parietal lesions. The use of prism glasses has been shown to substantially mitigate this condition.[97]
Attentional dyslexia
People with attentional dyslexia complain of letter-crowding or migration, sometimes blending elements of two words into one.[98] Sufferers read better when words are presented in isolation rather than flanked by other words and letters. Using a large magnifying glass may help mitigate this condition by reducing the effects of flanking from nearby words; however, no trials of this or indeed any other therapy for left parietal syndromes have been published as of 2014.[99]
Management
Main article: Management of dyslexia
Through the use of compensation strategies, therapy and educational support, dyslexic individuals can learn to read and write.[100] There are techniques and technical aids which help to manage or conceal symptoms of the disorder.[101] Removing stress and anxiety alone can sometimes improve written comprehension.[102] For dyslexia intervention with alphabet-writing systems, the fundamental aim is to increase a child's awareness of correspondences between graphemes (letters) and phonemes (sounds), and to relate these to reading and spelling by teaching how sounds blend into words. It has been found that reinforced collateral training focused on reading and spelling yields longer-lasting gains than oral phonological training alone.[103] Early intervention that is done for children at a young age can be successful in reducing reading failure.[104]
There is some evidence that the use of specially-tailored fonts may help with dyslexia.[1] These fonts, which include Dyslexie, OpenDyslexic, and Lexia Readable, were created based on the idea that many of the letters of the Latin alphabet are visually similar and may, therefore, confuse people with dyslexia. Dyslexie and OpenDyslexic both put emphasis on making each letter more distinctive in order to be more easily identified.[105] The benefits, however, might simply be due to the added spacing between words.[106]
There have been many studies conducted regarding intervention in dyslexia. Among these studies one meta-analysis found that there was functional activation as a result.[107]
There is no evidence demonstrating that the use of music education is effective in improving dyslexic adolescents' reading skills.[108]
Prognosis
Dyslexic children require special instruction for word analysis and spelling from an early age.[109] While there are fonts that may help people with dyslexia better understand writing, this might simply be due to the added spacing between words.[1][106] The prognosis, generally speaking, is positive for individuals who are identified in childhood and receive support from friends and family.[2]
Epidemiology
Map showing predominant forms of writing systems by country and what their characters represent:[110]
Alphabet (consonants and vowels)
Abjad (only consonants)
Abugida (family-related syllables)
Logograms
Syllabary (isolated syllables)
The percentage of people with dyslexia is unknown, but it has been estimated to be as low as 5% and as high as 17% of the population.[111] While it is diagnosed more often in males,[3] some believe that it affects males and females equally.
There are different definitions of dyslexia used throughout the world, but despite significant differences in writing systems, dyslexia occurs in different populations.[112] Dyslexia is not limited to difficulty in converting letters to sounds, and Chinese dyslexics may have difficulty converting Chinese characters into their meanings.[113][114] The Chinese vocabulary uses logographic, monographic, non-alphabet writing where one character can represent an individual phoneme.[115]
The phonological-processing hypothesis attempts to explain why dyslexia occurs in a wide variety of languages. Furthermore, the relationship between phonological capacity and reading appears to be influenced by orthography.[116]
History
Main article: History of developmental dyslexia
Dyslexia was identified by Oswald Berkhan in 1881,[44] but the term dyslexia was coined in 1887 by Rudolf Berlin, an ophthalmologist in Stuttgart.[117] He used the term to refer to the case of a young boy who had a severe impairment in learning to read and write, despite showing typical intelligence and physical abilities in all other respects.[118] In 1896, W. Pringle Morgan, a British physician from Seaford, East Sussex, published a description of a reading-specific learning disorder in a report to the British Medical Journal titled "Congenital Word Blindness".[119] The distinction between phonological and surface types of dyslexia is only descriptive, and without any etiological assumption as to the underlying brain mechanisms. However, studies have alluded to potential differences due to variation in performance.[120]
Research and society
Main article: Dyslexia research
See also: International Dyslexia Association
The majority of currently available dyslexia research relates to alphabetic writing systems, and especially to European languages.[121] However, substantial research is also available regarding dyslexics who speak Arabic, Chinese, Hebrew, or other languages.[122]
As is the case with any disorder, society often makes an assessment based on incomplete information. Before the 1980s, dyslexia was thought to be a consequence of education, rather than a basic disability. As a result, society often misjudges those with the disorder.[102] There is also sometimes a workplace stigma and negative attitude towards those with dyslexia.[123] If a dyslexic's instructors lack the necessary training to support a child with the condition, there is often a negative effect on the student's learning participation.[124]
See also
Child development
Linguistics
List of artistic depictions of dyslexia
Lists of language disorders
List of people diagnosed with dyslexia
Neurodiversity
Neurolinguistics
Philosophy of language
Elkonin boxes
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Further reading
Ramus, F; Altarelli, I; Jednoróg, K; Zhao, J; di Covella, LS (7 August 2017). "Neuroanatomy of developmental dyslexia: pitfalls and promise.". Neuroscience and biobehavioral reviews. ISSN 1873-7528. PMID 28797557. doi:10.1016/j.neubiorev.2017.08.001. Retrieved 31 August 2017. This article is published ahead of print
Alan Beaton (14 October 2004). Dyslexia, Reading and the Brain: A Sourcebook of Psychological and Biological Research. Psychology Press. ISBN 978-1-135-42275-2.
Thomas Richard Miles (4 August 2006). Fifty Years in Dyslexia Research. Wiley. ISBN 978-0-470-02747-9.
Gavin Reid; Angela Fawcett (12 May 2008). Dyslexia in Context: Research, Policy and Practice. John Wiley & Sons. ISBN 978-0-470-77801-2.
Michael Thomson (18 March 2009). The Psychology of Dyslexia: A Handbook for Teachers with Case Studies. John Wiley & Sons. ISBN 978-0-470-74197-9.
Gavin Reid (17 March 2011). Dyslexia (3 ed.). A&C Black. ISBN 978-1-4411-6585-5.
Mark Selikowitz (2 July 2012). Dyslexia and Other Learning Difficulties. Oxford University Press. ISBN 978-0-19-969177-7.
Andrew W. Ellis (25 February 2014). Reading, Writing and Dyslexia: A Cognitive Analysis. Psychology Press. ISBN 978-1-317-71630-3.
Julian G. Elliott; Elena L. Grigorenko (24 March 2014). The Dyslexia Debate. Cambridge University Press. ISBN 978-0-521-11986-3.
Agnew, Susie; Stewart, Jackie; Redgrave, Steve (8 October 2014). Dyslexia and Us: A collection of personal stories. Andrews UK Limited. ISBN 978-1-78333-250-2.
Norton, Elizabeth S.; Beach, Sara D.; Gabrieli, John D. E. (1 February 2015). "Neurobiology of dyslexia". Current Opinion in Neurobiology. 30: 73–78. ISSN 1873-6882. doi:10.1016/j.conb.2014.09.007. hdl:1721.1/102416.
Serrallach, Bettina; Groß, Christine; Bernhofs, Valdis; Engelmann, Dorte; Benner, Jan; Gündert, Nadine; Blatow, Maria; Wengenroth, Martina; Seitz, Angelika; Brunner, Monika; Seither, Stefan; Parncutt, Richard; Schneider, Peter; Seither-Preisler, Annemarie (15 July 2016). "Neural Biomarkers for Dyslexia, ADHD, and ADD in the Auditory Cortex of Children". Frontiers in Neuroscience. 10: 324. ISSN 1662-4548. PMC 4945653  . PMID 27471442. doi:10.3389/fnins.2016.00324.
Shao, Shanshan; Niu, Yanfeng; Zhang, Xiaohui; Kong, Rui; Wang, Jia; Liu, Lingfei; Luo, Xiu; Zhang, Jiajia; Song, Ranran (28 July 2016). "Opposite Associations between Individual KIAA0319 Polymorphisms and Developmental Dyslexia Risk across Populations: A Stratified Meta-Analysis by the Study Population". Scientific Reports. 6: 30454. Bibcode:2016NatSR...630454S. ISSN 2045-2322. PMC 4964335  . PMID 27464509. doi:10.1038/srep30454.
Brewer, Carmen C.; Zalewski, Christopher K.; King, Kelly A.; Zobay, Oliver; Riley, Alison; Ferguson, Melanie A.; Bird, Jonathan E.; McCabe, Margaret M.; Hood, Linda J.; Drayna, Dennis; Griffith, Andrew J.; Morell, Robert J.; Friedman, Thomas B.; Moore, David R. (11 October 2016). "Heritability of Non-Speech Auditory Processing Skills". European Journal of Human Genetics. 24 (8): 1137–1144. ISSN 1018-4813. PMC 4872837  . PMID 26883091. doi:10.1038/ejhg.2015.277.
Mascheretti, S.; De Luca, A.; Trezzi, V.; Peruzzo, D.; Nordio, A.; Marino, C.; Arrigoni, F. (3 January 2017). "Neurogenetics of developmental dyslexia: from genes to behavior through brain neuroimaging and cognitive and sensorial mechanisms". Translational Psychiatry. 7 (1): e987. PMID 28045463. doi:10.1038/tp.2016.240. Retrieved 8 January 2017.
Fraga González, Gorka; Žarić, Gojko; Tijms, Jurgen; Bonte, Milene; van der Molen, Maurits W. (18 January 2017). "Contributions of Letter-Speech Sound Learning and Visual Print Tuning to Reading Improvement: Evidence from Brain Potential and Dyslexia Training Studies". Brain Sciences. 7 (1): 10. PMC 5297299  . PMID 28106790. doi:10.3390/brainsci7010010.
Rudov, A; Rocchi, MB; Accorsi, A; Spada, G; Procopio, AD; Olivieri, F; Rippo, MR; Albertini, MC (October 2013). "Putative miRNAs for the diagnosis of dyslexia, dyspraxia, and specific language impairment.". Epigenetics. 8 (10): 1023–9. ISSN 1559-2308. PMID 23949389. doi:10.4161/epi.26026.
Vágvölgyi, R; Coldea, A; Dresler, T; Schrader, J; Nuerk, HC (2016). "A Review about Functional Illiteracy: Definition, Cognitive, Linguistic, and Numerical Aspects". Frontiers in psychology. 7: 1617. PMC 5102880  . PMID 27891100. doi:10.3389/fpsyg.2016.01617.
External links
Classification
V · T · D
ICD-10: F81.0 (developmental), R48.0
ICD-9-CM: 315.02
OMIM: 127700
MeSH: D004410
DiseasesDB: 4016
External resources
MedlinePlus: 001406
eMedicine: article/1835801
Patient UK: Dyslexia

Library resources about
Dyslexia
Resources in your library
Resources in other libraries
Wikimedia Commons has media related to Dyslexia.
Dyslexia
Misunderstood Minds Experience (simulating what dyslexics experience)
Topics related to Dyslexia
[show]
v
t
e
Dyslexia and related specific developmental disorders (F80–F83, 315)
General
conditions
Speech and
language
impairment,
communication
disorder
Expressive language disorder
Infantile speech
Landau–Kleffner syndrome
Language disorder
Lisp
Mixed receptive-expressive language disorder
Specific language impairment
Speech and language impairment
Speech disorder
Speech error
Speech sound disorder
Stammering
Tip of the tongue
Scholastic skills,
learning disorder
Developmental dyslexia
Dyscalculia
Dysgraphia (Disorder of written expression)
Motor function
Developmental coordination disorder
Developmental verbal dyspraxia also known as Childhood apraxia of speech
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Aphasias
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Conduction aphasia
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Neck mass
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Headache
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text:
Speed of the Sound of Loneliness
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"Speed of the Sound of Loneliness" is a song written by John Prine. It was released as a track on Prine's 1986 album German Afternoons.
The song gained further fame with a duet recording by Prine and American singer Nanci Griffith in the latter's 1993 album Other Voices, Other Rooms and the release of the track as a single.
The song has been covered by many, notably Kim Carnes who released it as a single from her 1988 album View from the House and by the English band Alabama 3 on their 1997 album Exile on Coldharbour Lane and American singer/songwriter Jeffrey Foucault in 2009 in his Prine tribute album Shoot the Moon Right Between the Eyes. Scottish singer Roddy Woomble recorded a live version in 2014 in his album Live at Kings Place.
[hide]
v
t
e
John Prine
Studio albums
John Prine (1971)
Diamonds in the Rough (1972)
Sweet Revenge (1973)
Common Sense (1975)
Bruised Orange (1978)
Pink Cadillac (1979)
Storm Windows (1980)
Aimless Love (1984)
German Afternoons (1986)
The Missing Years (1991)
A John Prine Christmas (1994)
Lost Dogs and Mixed Blessings (1995)
In Spite of Ourselves (1999)
Souvenirs (2000)
Fair & Square (2005)
Standard Songs for Average People (with Mac Wiseman) (2007)
For Better, or Worse (2016)
Live albums
John Prine Live (1988)
Live on Tour (1997)
In Person & On Stage (2010)
Compilation albums
Prime Prine: The Best of John Prine (1976)
Great Days: The John Prine Anthology (1993)
Songs
"Sam Stone"
"Paradise"
"Your Flag Decal Won't Get You Into Heaven Anymore"
"Angel from Montgomery"
"The Great Compromise"
"Speed of the Sound of Loneliness"
Related
Oh Boy Records
John Prine - Live from Sessions at West 54th
Broken Hearts & Dirty Windows: Songs of John Prine
Retrieved from "https://en.wikipedia.org/w/index.php?title=Speed_of_the_Sound_of_Loneliness&oldid=777788786"
Categories:
1986 songs
1993 singles
John Prine songs
Nanci Griffith songs
Songs written by John Prine
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This page was last edited on 29 April 2017, at 07:53.
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